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Editorial commentary: The air that I breathe……………..makes me sick!.

Trends in cardiovascular medicine

Münzel T.
PMID: 28967463
Trends Cardiovasc Med. 2018 Feb;28(2):127-129. doi: 10.1016/j.tcm.2017.08.014. Epub 2017 Aug 30.

No abstract available.

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Münzel T. Editorial commentary: The air that I breathe……………..makes me sick!. Trends Cardiovasc Med. 2017;28(2):127-129doi: 10.1016/j.tcm.2017.08.014.
Münzel, T. (2018). Editorial commentary: The air that I breathe……………..makes me sick!. Trends in cardiovascular medicine, 28(2), 127-129. https://doi.org/10.1016/j.tcm.2017.08.014
Münzel, Thomas. "Editorial commentary: The air that I breathe……………..makes me sick!." Trends in cardiovascular medicine vol. 28,2 (2018): 127-129. doi: https://doi.org/10.1016/j.tcm.2017.08.014
Münzel T. Editorial commentary: The air that I breathe……………..makes me sick!. Trends Cardiovasc Med. 2018 Feb;28(2):127-129. doi: 10.1016/j.tcm.2017.08.014. Epub 2017 Aug 30. PMID: 28967463.
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Do genes change the matter?.

Trends in cardiovascular medicine

Postema PG.
PMID: 26344599
Trends Cardiovasc Med. 2016 Apr;26(3):266-7. doi: 10.1016/j.tcm.2015.08.001. Epub 2015 Aug 10.

No abstract available.

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Postema PG. Do genes change the matter?. Trends Cardiovasc Med. 2015;26(3):266-7doi: 10.1016/j.tcm.2015.08.001.
Postema, P. G. (2016). Do genes change the matter?. Trends in cardiovascular medicine, 26(3), 266-7. https://doi.org/10.1016/j.tcm.2015.08.001
Postema, Pieter G. "Do genes change the matter?." Trends in cardiovascular medicine vol. 26,3 (2016): 266-7. doi: https://doi.org/10.1016/j.tcm.2015.08.001
Postema PG. Do genes change the matter?. Trends Cardiovasc Med. 2016 Apr;26(3):266-7. doi: 10.1016/j.tcm.2015.08.001. Epub 2015 Aug 10. PMID: 26344599.
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Universal therapeutic targeting of age-related protein quality control system dysfunction in chronic diseases?.

Trends in cardiovascular medicine

Van Eyk JE.
PMID: 25802225
Trends Cardiovasc Med. 2015 Apr;25(3):248-9. doi: 10.1016/j.tcm.2015.01.012. Epub 2015 Feb 07.

No abstract available.

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Van Eyk JE. Universal therapeutic targeting of age-related protein quality control system dysfunction in chronic diseases?. Trends Cardiovasc Med. 2015;25(3):248-9doi: 10.1016/j.tcm.2015.01.012.
Van Eyk, J. E. (2015). Universal therapeutic targeting of age-related protein quality control system dysfunction in chronic diseases?. Trends in cardiovascular medicine, 25(3), 248-9. https://doi.org/10.1016/j.tcm.2015.01.012
Van Eyk, Jennifer E. "Universal therapeutic targeting of age-related protein quality control system dysfunction in chronic diseases?." Trends in cardiovascular medicine vol. 25,3 (2015): 248-9. doi: https://doi.org/10.1016/j.tcm.2015.01.012
Van Eyk JE. Universal therapeutic targeting of age-related protein quality control system dysfunction in chronic diseases?. Trends Cardiovasc Med. 2015 Apr;25(3):248-9. doi: 10.1016/j.tcm.2015.01.012. Epub 2015 Feb 07. PMID: 25802225.
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Editorial Commentary: The metabolic syndrome: What does the clinician need to know and do?.

Trends in cardiovascular medicine

Thompson PD.
PMID: 27013136
Trends Cardiovasc Med. 2016 May;26(4):374-5. doi: 10.1016/j.tcm.2015.11.001. Epub 2015 Nov 19.

No abstract available.

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Thompson PD. Editorial Commentary: The metabolic syndrome: What does the clinician need to know and do?. Trends Cardiovasc Med. 2015;26(4):374-5doi: 10.1016/j.tcm.2015.11.001.
Thompson, P. D. (2016). Editorial Commentary: The metabolic syndrome: What does the clinician need to know and do?. Trends in cardiovascular medicine, 26(4), 374-5. https://doi.org/10.1016/j.tcm.2015.11.001
Thompson, Paul D. "Editorial Commentary: The metabolic syndrome: What does the clinician need to know and do?." Trends in cardiovascular medicine vol. 26,4 (2016): 374-5. doi: https://doi.org/10.1016/j.tcm.2015.11.001
Thompson PD. Editorial Commentary: The metabolic syndrome: What does the clinician need to know and do?. Trends Cardiovasc Med. 2016 May;26(4):374-5. doi: 10.1016/j.tcm.2015.11.001. Epub 2015 Nov 19. PMID: 27013136.
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Editorial Commentary: Bringing precision medicine to intervention: Virtually a reality.

Trends in cardiovascular medicine

Majithia A, Bhatt DL.
PMID: 27068765
Trends Cardiovasc Med. 2016 Jul;26(5):474-6. doi: 10.1016/j.tcm.2016.03.002. Epub 2016 Mar 09.

No abstract available.

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Majithia A, Bhatt DL. Editorial Commentary: Bringing precision medicine to intervention: Virtually a reality. Trends Cardiovasc Med. 2016;26(5):474-6doi: 10.1016/j.tcm.2016.03.002.
Majithia, A., & Bhatt, D. L. (2016). Editorial Commentary: Bringing precision medicine to intervention: Virtually a reality. Trends in cardiovascular medicine, 26(5), 474-6. https://doi.org/10.1016/j.tcm.2016.03.002
Majithia, Arjun, and Bhatt, Deepak L. "Editorial Commentary: Bringing precision medicine to intervention: Virtually a reality." Trends in cardiovascular medicine vol. 26,5 (2016): 474-6. doi: https://doi.org/10.1016/j.tcm.2016.03.002
Majithia A, Bhatt DL. Editorial Commentary: Bringing precision medicine to intervention: Virtually a reality. Trends Cardiovasc Med. 2016 Jul;26(5):474-6. doi: 10.1016/j.tcm.2016.03.002. Epub 2016 Mar 09. PMID: 27068765.
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Anticoagulant thrombins.

Trends in cardiovascular medicine

Di Cera E.
PMID: 14987548
Trends Cardiovasc Med. 1998 Nov;8(8):340-50. doi: 10.1016/s1050-1738(98)00030-9.

Thrombin plays both procoagulant and anticoagulant roles in the blood coagulation cascade. This dual role is influenced allosterically by the binding of Na(+) near the primary specificity pocket of the enzyme. Recent findings demonstrate that it is possible to...

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Di Cera E. Anticoagulant thrombins. Trends Cardiovasc Med. 1998;8(8):340-50doi: 10.1016/s1050-1738(98)00030-9.
Di Cera, E. (1998). Anticoagulant thrombins. Trends in cardiovascular medicine, 8(8), 340-50. https://doi.org/10.1016/s1050-1738(98)00030-9
Di Cera, E. "Anticoagulant thrombins." Trends in cardiovascular medicine vol. 8,8 (1998): 340-50. doi: https://doi.org/10.1016/s1050-1738(98)00030-9
Di Cera E. Anticoagulant thrombins. Trends Cardiovasc Med. 1998 Nov;8(8):340-50. doi: 10.1016/s1050-1738(98)00030-9. PMID: 14987548.
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Allosteric regulation of the cofactor-dependent serine protease coagulation factor VIIa.

Trends in cardiovascular medicine

Ruf W, Dickinson CD.
PMID: 14987549
Trends Cardiovasc Med. 1998 Nov;8(8):350-6. doi: 10.1016/s1050-1738(98)00031-0.

The integration of structure and function analysis of the tissue factor-factor VIIa complex has provided a detailed view of the functional surface of the extrinsic activation complex. An incomplete zymogen to enzyme transition is responsible for the strict cofactor...

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Ruf W, Dickinson CD. Allosteric regulation of the cofactor-dependent serine protease coagulation factor VIIa. Trends Cardiovasc Med. 1998;8(8):350-6doi: 10.1016/s1050-1738(98)00031-0.
Ruf, W., & Dickinson, C. D. (1998). Allosteric regulation of the cofactor-dependent serine protease coagulation factor VIIa. Trends in cardiovascular medicine, 8(8), 350-6. https://doi.org/10.1016/s1050-1738(98)00031-0
Ruf, W, and Dickinson, C D. "Allosteric regulation of the cofactor-dependent serine protease coagulation factor VIIa." Trends in cardiovascular medicine vol. 8,8 (1998): 350-6. doi: https://doi.org/10.1016/s1050-1738(98)00031-0
Ruf W, Dickinson CD. Allosteric regulation of the cofactor-dependent serine protease coagulation factor VIIa. Trends Cardiovasc Med. 1998 Nov;8(8):350-6. doi: 10.1016/s1050-1738(98)00031-0. PMID: 14987549.
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Fibronectin-fibrin cross-linking: a regulator of cell behavior.

Trends in cardiovascular medicine

Corbett SA, Schwarzbauer JE.
PMID: 14987550
Trends Cardiovasc Med. 1998 Nov;8(8):357-62. doi: 10.1016/s1050-1738(98)00028-0.

At sites of tissue injury or inflammation, extravasation of plasma proteins leads to the formation of a complex fibrillar matrix composed primarily of fibrin and plasma fibronectin (pFN). This protein meshwork serves not only to reestablish the integrity of...

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Corbett SA, Schwarzbauer JE. Fibronectin-fibrin cross-linking: a regulator of cell behavior. Trends Cardiovasc Med. 1998;8(8):357-62doi: 10.1016/s1050-1738(98)00028-0.
Corbett, S. A., & Schwarzbauer, J. E. (1998). Fibronectin-fibrin cross-linking: a regulator of cell behavior. Trends in cardiovascular medicine, 8(8), 357-62. https://doi.org/10.1016/s1050-1738(98)00028-0
Corbett, S A, and Schwarzbauer, J E. "Fibronectin-fibrin cross-linking: a regulator of cell behavior." Trends in cardiovascular medicine vol. 8,8 (1998): 357-62. doi: https://doi.org/10.1016/s1050-1738(98)00028-0
Corbett SA, Schwarzbauer JE. Fibronectin-fibrin cross-linking: a regulator of cell behavior. Trends Cardiovasc Med. 1998 Nov;8(8):357-62. doi: 10.1016/s1050-1738(98)00028-0. PMID: 14987550.
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Molecular basis of vascular anomalies.

Trends in cardiovascular medicine

Vikkula M, Boon LM, Mulliken JB, Olsen BR.
PMID: 14987552
Trends Cardiovasc Med. 1998 Oct;8(7):281-92. doi: 10.1016/s1050-1738(98)00024-3.

Vascular anomalies comprise a heterogeneous group of disorders that are divided into tumors (hemangiomas) and malformations. Recent advances in biomedical research provide insights into the molecular basis of these disorders and a deeper understanding of vascular morphogenesis. In the...

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Vikkula M, Boon LM, Mulliken JB, et al. Molecular basis of vascular anomalies. Trends Cardiovasc Med. 1998;8(7):281-92doi: 10.1016/s1050-1738(98)00024-3.
Vikkula, M., Boon, L. M., Mulliken, J. B., & Olsen, B. R. (1998). Molecular basis of vascular anomalies. Trends in cardiovascular medicine, 8(7), 281-92. https://doi.org/10.1016/s1050-1738(98)00024-3
Vikkula, M, et al. "Molecular basis of vascular anomalies." Trends in cardiovascular medicine vol. 8,7 (1998): 281-92. doi: https://doi.org/10.1016/s1050-1738(98)00024-3
Vikkula M, Boon LM, Mulliken JB, Olsen BR. Molecular basis of vascular anomalies. Trends Cardiovasc Med. 1998 Oct;8(7):281-92. doi: 10.1016/s1050-1738(98)00024-3. PMID: 14987552.
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Functional interactions of cytoplasmic domains of the skeletal muscle Ca2+ release channel.

Trends in cardiovascular medicine

Wu Y, Hamilton SL.
PMID: 14987556
Trends Cardiovasc Med. 1998 Oct;8(7):312-9. doi: 10.1016/s1050-1738(98)00023-1.

The skeletal muscle Ca(2+) release channel (RYR1) is a homotetramer with subunits of 565 kD. Although the channel part of this protein is probably formed by the C-terminal one fifth of the protein, most of the regulation of channel...

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Wu Y, Hamilton SL. Functional interactions of cytoplasmic domains of the skeletal muscle Ca2+ release channel. Trends Cardiovasc Med. 1998;8(7):312-9doi: 10.1016/s1050-1738(98)00023-1.
Wu, Y., & Hamilton, S. L. (1998). Functional interactions of cytoplasmic domains of the skeletal muscle Ca2+ release channel. Trends in cardiovascular medicine, 8(7), 312-9. https://doi.org/10.1016/s1050-1738(98)00023-1
Wu, Y, and Hamilton, S L. "Functional interactions of cytoplasmic domains of the skeletal muscle Ca2+ release channel." Trends in cardiovascular medicine vol. 8,7 (1998): 312-9. doi: https://doi.org/10.1016/s1050-1738(98)00023-1
Wu Y, Hamilton SL. Functional interactions of cytoplasmic domains of the skeletal muscle Ca2+ release channel. Trends Cardiovasc Med. 1998 Oct;8(7):312-9. doi: 10.1016/s1050-1738(98)00023-1. PMID: 14987556.
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The mechanisms by which infectious agents may contribute to atherosclerosis and its clinical manifestations.

Trends in cardiovascular medicine

Kol A, Libby P.
PMID: 14987564
Trends Cardiovasc Med. 1998 Jul;8(5):191-9. doi: 10.1016/s1050-1738(98)00010-3.

Three infectious agents have recently gained considerable interest as potential pathogens in atherosclerosis and in its clinical manifestations: herpes simplex virus, cytomegalovirus, and Chlamydia pneumoniae. Chronic and often asymptomatic infections with these agents occur widely in the general population....

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Kol A, Libby P. The mechanisms by which infectious agents may contribute to atherosclerosis and its clinical manifestations. Trends Cardiovasc Med. 1998;8(5):191-9doi: 10.1016/s1050-1738(98)00010-3.
Kol, A., & Libby, P. (1998). The mechanisms by which infectious agents may contribute to atherosclerosis and its clinical manifestations. Trends in cardiovascular medicine, 8(5), 191-9. https://doi.org/10.1016/s1050-1738(98)00010-3
Kol, A, and Libby, P. "The mechanisms by which infectious agents may contribute to atherosclerosis and its clinical manifestations." Trends in cardiovascular medicine vol. 8,5 (1998): 191-9. doi: https://doi.org/10.1016/s1050-1738(98)00010-3
Kol A, Libby P. The mechanisms by which infectious agents may contribute to atherosclerosis and its clinical manifestations. Trends Cardiovasc Med. 1998 Jul;8(5):191-9. doi: 10.1016/s1050-1738(98)00010-3. PMID: 14987564.
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Auxiliary subunits of Shaker-type potassium channels.

Trends in cardiovascular medicine

Xu J, Li M.
PMID: 14987569
Trends Cardiovasc Med. 1998 Jul;8(5):229-34. doi: 10.1016/s1050-1738(98)00011-5.

Voltage-gated potassium channels are important determinants of membrane excitability. This family of ion channels is composed of several classes of proteins, including the pore-forming Kvalpha subunits and the recently identified auxiliary Kvbeta subunits. A combination of a large number...

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Xu J, Li M. Auxiliary subunits of Shaker-type potassium channels. Trends Cardiovasc Med. 1998;8(5):229-34doi: 10.1016/s1050-1738(98)00011-5.
Xu, J., & Li, M. (1998). Auxiliary subunits of Shaker-type potassium channels. Trends in cardiovascular medicine, 8(5), 229-34. https://doi.org/10.1016/s1050-1738(98)00011-5
Xu, J, and Li, M. "Auxiliary subunits of Shaker-type potassium channels." Trends in cardiovascular medicine vol. 8,5 (1998): 229-34. doi: https://doi.org/10.1016/s1050-1738(98)00011-5
Xu J, Li M. Auxiliary subunits of Shaker-type potassium channels. Trends Cardiovasc Med. 1998 Jul;8(5):229-34. doi: 10.1016/s1050-1738(98)00011-5. PMID: 14987569.
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Showing 1 to 12 of 599 entries